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Mtb-induced macrophage death was related to a decrease in GSH and GPX4 levels and an increase in free iron, mitochondrial superoxide, and lipid peroxidation, which indicates that ferroptosis was involved in tissue necrosis in Mtb infection (Yang et al., 2022)
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Framing it accurately helps set realistic expectations: glutathione replenishment supports the body's own cellular defense systems
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