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The symptoms are stimulated by the release of toxic bacterial components into the bloodstream when the bacteria break apart 3 4 5
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In fact, binding could be induced either by hydrophobic interactions between allyl chain and hydrophobic regions of A β (Phe 19 and Val 12), or by the H-bond between the -OH group of the carboxylic group of SAC and donator/acceptor groups of A β [102]
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