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[DOI] [PubMed] [Google Scholar] 28.Bengtsson BA, Eden S, Lonn L, Kvist H, Stokland A, Lindstedt G, Bosaeus I, Tolli J, Sjostrom L, Isaksson OGP
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however, its exact contribution to brain Se transport was not systematically studied and it seems to be rather limited since megalin –/– mice do not exhibit neurological phenotype associated with Se deficiency (Kurokawa et al., 2014b), typical for Selenop –/– or Lrp8 –/– mice
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