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Hu Z, Li F, Hu C, et al
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This mechanism enables: Enhanced cellular replicative potential beyond the Hayflick limit Restoration of telomere length in aging cell populations Improved cellular survival under oxidative stress conditions Activation of genes associated with cellular repair and maintenance Studies using human fetal fibroblasts revealed that Epithalon treatment extended cell proliferation from 34 passages in controls to beyond 44 passages in treated cells, with telomere lengths returning to levels comparable to early-passage cells[2]
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