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Neutrophils bearing XKR8 2DA mutations exhibited a marked defect in PS exposure upon PMA stimulation and during apoptosis, closely resembling the phenotype observed in XKR8 KO neutrophils (Fig
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Modulating NF-κB, MAPK, and PI3K/AKT signaling by ergothioneine attenuates iron overload-induced hepatocellular injury in rats
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