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It involves the synthesis and repair of DNA and RNA, as well as the regulation of the cell cycle, thereby impacting the pathogenesis of gliomas from multiple angles [154]

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When the skin is injured, the balance of the transmembrane potential is disrupted, and the combination of cellular damage, ion channel opening, and inflammation after the injury leads to a decrease in the potential at the wound site, while the TEP around the wound remains unchanged, forming a low-resistance channel, resulting in a “short-circuitat the wound site”, and thus, the TEP drives the electric current flows out of the short-circuit site that occurs at the skin injury (Tai et al., 2009)