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While abstinence remains the cornerstone intervention for alcohol-associated liver disease (ALD), the refractory nature and high relapse propensity of AUD necessitate deeper investigations into fundamental regulatory mechanisms [14]

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Research has demonstrated that OMT protects ECs from Hcy‐induced damage by inhibiting MMPs expression, increasing the Bcl‐2/Bax ratio, and suppressing the expression of caspase‐3 and caspase‐9