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Application of ADDLs to cultured neurons or slices has been found to lead to a significant loss of dendritic spines 175–177

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During the development of nonalcoholic fatty liver disease (NAFLD), disorders of the carnitine pool are considered important drivers, in which the LCAC profile changes significantly with the progression of NAFLD and provides key clues for revealing the complex pathogenesis of NAFLD [209, 210]

Carnitine is known for its ability to assist in the transport of fatty acids into the mitochondria