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Consequently, these inabilities of growth factors are practical obstacles that could preclude suited determination of the underlying general mechanisms that could back the obtained findings (for review see [14])

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In human RBCs, G6PD deficiency causes sulfhydryl group oxidation, conversion of GSH to GSSG, decreased activity of GPX1 and PRDX2, and displacement of cell redox balance to an oxidized state [75,76]

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