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Alternative inflammatory pathways involving gasdermin E and caspase-8/3 enable persistent cytokine release in the absence of gasdermin D, revealing redundant effector arms within the inflammasome network

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Targeting M2phenotypic TAMs reduces the polarization of macrophages toward the M2 phenotype and raises the amount of M1-TAMs, which in turn strengthens the anti-tumor immune microenvironment (38)