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Of note, Group I mGluRs (mGluR 1 and mGluR 5 ) expressed on glutamatergic synapses localized in the spinal cord of SOD1 G93A ALS mice resulted in overexpression and enhanced sensitivity to the agonist present in the synaptic cleft
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The recent revelation that the velocity storage mechanism may be involved in MdDS opened opportunities for positive long-term outcomes by targeting the root cause of the illness under a unified vestibular system-based perspective (5, 22–25)
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